Skip to main content

Table 1 Tissue characteristics in controls and patient groups

From: Native T1-mapping detects the location, extent and patterns of acute myocarditis without the need for gadolinium contrast agents

CMR findings

Controls (n = 50)

All patients (n = 60)

Group I (Edema+, LGE+) (n = 41)

Group II (Edema-, LGE+) (n = 12)

Group III (Edema-, LGE-) (n = 7)

Ejection fraction (%)

72 ± 6

64 ± 12*

61 ± 13†

70 ± 11

72 ± 6

Global myocardial T2 SI ratio‡

1.56 ± 0.15

1.71 ± 0.27*

1.79 ± 0.27†

1.60 ± 0.16

1.47 ± 0.17

Myocardial injury by T2 SI ratio ≥ 2.0 (%)

0 (0 to 2)

5 (1 to 18)*

9 (3 to 22)†

0.6 (0 to 4)

0 (0 to 0)

Mean myocardial T1 (ms)

946 ± 23

1011 ± 64*

1030 ± 62†

986 ± 54†

947 ± 27

Myocardial injury by T1 ≥ 990 ms (%)

0.7 (0 to 3)

32 (15 to 65)*

49 (24 to 70)†

15 (12 to 29)†

6 (2 to 7)†

Myocardial injury by LGE (%)§

0 (0 to 1) (n = 35)

11 (4 to 20)*

16 (8 to 24)†

7 (4 to 12)†

1 (0 to 2)

  1. Values are presented as mean ± SD or median (interquartile range). *p < 0.001 when compared to controls.
  2. In post-hoc subgroup analyses: †significantly different from controls;
  3. areas of injury by T2 (edema) were significantly larger in Group I than Group II and III;
  4. areas of injury by both T1 and LGE were significantly larger in Group I > II > III patients.
  5. ‡Global myocardial T2 SI ratio = T2 SI myocardium: sk. muscle
  6. §LGE is detected when myocardial SI is ≥ 2.0 standard deviation above the mean SI of remote myocardium.