Fig. 2From: The predictive capabilities of a novel cardiovascular magnetic resonance derived marker of cardiopulmonary reserve on established prognostic surrogate markers in patients with pulmonary vascular disease: results of a longitudinal pilot studyChanges in RHC-derived hemodynamics (panel 1) and CMR-derived meanPAvel (initial assessment, panel 2a; follow-up assessment, panel 2b) in response to adenosine in PAH and High Risk participants. At RHC, intravenous adenosine produced a dose dependent reduction in PVRI driven predominantly by a higher CI rather than a lower mPAP or transpulmonary gradient (not illustrated). At CMR, these changes were reflected by a dose-dependent rise in meanPAvel. Changes were more pronounced in High Risk participants relative to those with PAHBack to article page