Fig. 5
Comparison between the proposed 3D whole-heart BOOST framework and the clinical 2D PSIR acquisition. Images in a, e, and i show the LGE uptake as depicted in the T2Prep-IR BOOST datasets (white arrows), where signal from the blood pool is present and the viable myocardium is suppressed. Reformats in b, f, and j show the coronary reformats obtained from the 3D whole-heart bright-blood T2Prep BOOST dataset. Complementary 3D black-blood LGE images obtained with BOOST are shown in c, g, and k. All the images from the T2Prep-IR BOOST and the PSIR BOOST datasets were reformatted to match the orientation of the clinical 2D PSIR acquisitions (d, h, and l). The LGE uptake identified in both the T2Prep-IR BOOST and PSIR BOOST datasets matches that of the clinical 2D PSIR acquisition. Furthermore, improved contrast between the scar tissue and the blood pool can be appreciated in the 3D PSIR BOOST datasets when compared to the 2D PSIR acquisition (g, k versus h, l, orange arrows). LGE uptake appears more shallow and blood pool signal is not entirely suppressed in Patient 04 with myocarditis (c, d, orange arrows), due to a longer TI