Figure 5

Example CMR findings. Midventricular short axis T2-weighted short tau inversion recovery (STIR), late gadolinium enhancement (LGE), and T2 mapping sequences in a 29-year-old healthy male volunteer (A, B, C), a 42-year-old patient (#1) with acute, diffuse myocarditis (D, E, F), and a 53-year-old patient (#2) with acute myocardial infarction (G, H, I). Mean global T2 relaxation time for the healthy volunteer was 48.5 ± 4.6 ms (anterior: 47.8 ms; anteroseptal: 57.8 ms; inferoseptal: 52.1 ms; inferior: 49.6 ms; inferolateral: 45.5 ms; anterolateral: 47.1 ms) (C). In patient #1 diffuse myocardial edema was present throughout all myocardial segments (D, E, F), associated with increased global T2 values of 68.0 ± 2.4 ms (anterior: 65.4 ms; anteroseptal: 68.4 ms; inferoseptal: 70.5 ms; inferior: 65.8 ms; inferolateral: 64.4 ms; anterolateral: 66.1 ms). Patient #2 showed an acute myocardial infarction of the inferior segments with subendocardial LGE (G) and pronounced edema in the T2 STIR image (H). Pronounced myocardial edema is also visible in the corresponding T2 map (I), where the infarcted segments showed mean T2 relaxation times of 89.4 ms (inferior) and 82.3 ms (inferolateral) (anterior: 55.5 ms; anteroseptal: 56.2 ms; inferoseptal: 70.5 ms; anterolateral: 66.1 ms; global T2 relaxation time: 62.5 ± 13.1 ms).