Fig. 2

Flowchart outlining patient selection for the study. DCRV – double-chambered right ventricle, CABG – coronary artery bypass grafting. *A definitive diagnosis causing LV hypertrophy other than HCM or imitating HCM included the following: cardiac amyloidosis, Fabry disease, aortic stenosis, eosinophilic syndrome, and previous myocardial infarction with LV wall thinning and LV hypertrophy of the viable myocardium. ^#Included 13 patients after alcohol septal ablation (one patient with subsequent endocardial radiofrequency ablation of septal hypertrophy), one patient with previous percutaneous transluminal septal coil embolization, and seven patients after surgical myectomy (three patients had previous alcohol septal ablation). ^§Artifacts due to frequent premature ventricular or supraventricular contractions, claustrophobia, anxiety, post-stroke aphasia, and vertebral column stabilization implants. ^†Included a history of atrial septal defect closure/intraatrial septum shunt, third-degree atrioventricular block, presence of left ventricular thrombus, and a significant amount of pericardial effusion/history of pericarditis. ^‡Included patients with coexistence of HCM and severe hypertension; patients with papillary muscle abnormalities and/or prominent myocardial crypts without overt LV hypertrophy; and a proband or family member with borderline LV wall thickness that required further genetic testing. ^¶Included family members of a patient with HCM studied as part of a screening program, patients with no LV hypertrophy present with a diagnosis of overestimation of ventricular wall measurements with the use of echocardiography, and patients with athlete’s heart